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If you want to start taking any new medicine or supplement while you are taking imatinib, talk to your doctor first.

In patients with CML, nausea, vomiting, diarrhea, edema, muscle cramps, musculoskeletal pain, rash.

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ULN occur in 1-3% of patients on imatinib alone and in 53% of those receiving imatinib in combination with other agents; 5 reports of liver injury have been published including one case of acute liver failure).

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There is evidence that MCAD is a disorder with considerable prevalence and thus should be considered routinely in the differential diagnosis of patients with chronic multisystem polymorbidity of unknown cause.

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Bilateral Toxoplasma Retinochoroiditis in a Patient with Chronic Myeloid Leukemia Treated with Imatinib Mesylate.

Complete cytogenetic responses occurred in more than 40 percent of patients with chronic-phase CML, a higher rate than that associated with interferon therapy.

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Changing the cost of care for chronic myeloid leukemia: the availability of generic imatinib in the USA and the EU.

Bone marrow morphology and cytogenetics were evaluated every 12 weeks.

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This might be a result of the fact that the drugs are taken up in cells by different mechanisms: imatinib influx is dependent on OCT1 but nilotinib is not.

Furthermore, we recently reported that sodium selenite induces mitophagic cell death (cell death by excessive mitophagy) selectively in malignant glioma cells, but not in normal astrocytes (; ), suggesting that selenite may prove useful for cancer therapy via a mitochondria-selective type of autophagic cell death.

As illustrated in the above figure, patients who achieved partial response and those with stable disease show very similar durations of survival in the US-Finland study.

This risk may be higher if you take cyclosporine or cyclosporine (modified) with other medications that decrease the functioning of the immune system such as azathioprine (Imuran), cancer chemotherapy, methotrexate (Rheumatrex), (), and tacrolimus (Prograf).

Trent has published extensively about GIST and other sarcomas and is quite active in clinical trials.

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Department of Surgery, Wayne State University School of Medicine, Detroit, MI.

Continued treatment with imatinib resulted in further reductions in minimal residual disease, but few, if any, responding patients actually become PCR negative for BCR-ABL transcripts during imatinib therapy.

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The most parsimonious explanation for these findings is that imatinib directly stimulates bone formation while restraining resorption ().

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The cause for the condition itself is unknown, and the diagnosis is made when other health conditions are ruled out.

Potent antitumor activities of recombinant human PDCD5 protein in combination with chemotherapy drugs in K562 cells.

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Imatinib was reduced to 600 and 400 mg daily, 9 and 12 months after transplantation, respectively, and discontinued 15 months after transplantation.

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Specific therapy for the hyperthyroid state might lead to a moderate increase of the platelet count.

This test is expensive because it is based on years of proprietary genetic research and multiple large-scale clinical trials to confirm its predictive capability.

However, in a review of a large number of patients treated on Children's Oncology Group (COG) trials over a 15-year period, T-cell immunophenotype still proved to be a negative prognostic factor on multivariate analysis.

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None of the patients however was strictly in CMR throughout the entire 2-year period preceding cessation of imatinib and half of the patients were high risk according to the Sokal index.

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Impact of BCR-ABL transcript type on outcome in patients with chronic-phase CML treated with tyrosine kinase inhibitors.

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NCI-funded clinical trials as well as studies conducted by investigators at hospitals and medical centers throughout the United States and around the world.

The intracardiac shunt leads to hypoxia and a resultant secondary polycythemia.

Effect of interaction of glutathione S-transferases (T1 and M1) on the hematologic and cytogenetic responses in chronic myeloid leukemia patients treated with imatinib.

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One possible explanation could be the inhibition of targets that are responsible for the integrity of capillaries by imatinib.

This is the first data-based estimation of the expectable survival benefits for chronic phase CML patients treated with imatinib.

ALL suggested that the adverse prognostic significance of this subtype may be abrogated when patients are treated with risk-directed therapy based on MRD levels.

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CML) who no longer benefit from or did not tolerate other treatment.

In these studies, IFN therapy not only delayed the time of disease progression but also prolonged overall survival.

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Hematological responses to minimal dose of imatinib in this cohort (300 mg daily) were obtained in 55% and 70% of patients with myeloid, and lymphoid blast crisis respectively, including four CHR in each group.

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Following the performance of this analysis, two additional events occurred on the nilotinib 400 mg BID arm between 3 and 4 years; these events were not included in the current statistical analysis.

Still more preferably, the procedure of the invention is conducted by preparing a suspension of Imatinib mesylate in n-Butanol.

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FU therapy increases the risk of thiamine deficiency, and hence WE, as it blocks the conversion of thiamine to its active metabolite thiamine pyrophosphate (TPP).

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She was treated successfully with multiple platelet and blood transfusions, along with cyclosporine, antithymocyte globulin (ATG), and corticosteroids.

Prospective clinical trials have supported the efficacy and safety of imatinib before and after surgical resection of GIST.

These effects may be worse if you take it with alcohol or certain medicines.

Preclinical characterization of GLPG0634, a selective inhibitor of JAK1 for the treatment of inflammatory diseases".

Studies on treatment of acute promyelocytic leukemia with arsenic trioxide: remission induction, follow-up, and molecular monitoring in 11 newly diagnosed and 47 relapsed acute promyelocytic leukemia patients.

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Gleevec is the trade name for the generic drug name Imatinib Mesylate.

Outcome of children with relapsed acute myeloid leukemia following initial therapy under the AML99 protocol.

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The Abl kinase inhibitor imatinib was registered for clinical use in 2001 and is now the current first-line treatment for CML.

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Detection of Protein Tyrosine-Kinase (PTK) Gene Expression Pattern in Normal and Malignant T Lymphocytes by Combined PTK-Specific Polymerase Chain Reaction and Parallel Denaturing Gradient Gel Electrophoresis.

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Expression of the BCR-ABL gene is sufficient to cause chronic phase CML, while disease progression to AP or BC is thought to depend on additional genetic changes.

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Nilotinib versus imatinib for newly diagnosed chronic-phase chronic myeloid leukemia.

Evaluation of the safety and efficacy of an oral molecularly-targeted therapy, STI571, in patients (Pts) with unresectable or metastatic gastrointestinal stromal tumors (GISTs) expressing c-kit (CD117).

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In the Associazione Italiana Ematologia Oncologia Pediatrica (AIEOP) trial, MRD status at day 78 (week 12) was the most important predictor for relapse in patients with T-cell ALL.

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Diagnosis of gastrointestinal tract lesions by endoscopic ultrasound-guided fine-needle aspiration biopsy.

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Reduced ABCG2 and increased SLC22A1 mRNA expression are associated with imatinib response in chronic myeloid leukemia.

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Congestive heart failure (impaired ability of the heart to pump blood) and left ventricular dysfunction (impaired functioning of the left side of the heart) have been reported, particularly in patients with other health issues and risk factors.

Acid phosphatase activity was used to measure LX-2 cell viability following treatment with CBD and imatinib as in (b).

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Median survival for imatinib-treated people with (GIST) is nearly 5 years compared to 9 to 20 months in the pre-imatinib-era.

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In summary, pleural effusion is a complication of dasatinib therapy in CML.

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Given this premise, CML patients with a more complex karyotype were expected to be more resistant to imatinib mesylate.

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